Module Special Admissions Requirements
Study of Level 1 Cell Biology
Module Texts
Suggested resources to support the module are:
D.L. Nelson & M.M. Cox (2008) Lehninger Principles of Biochemistry 5th Revised edition. W.H. Freeman & Co Ltd with on-line access available from John Smiths Bookshop in the University Library
or
D. L. Nelson & M. M. Cox (2008) Lehninger Principles of Biochemistry 5th Revised edition. W.H. Freeman & Co Ltd ISBN 9781429208925
Berg, J.M., Tymoczko, J.L. and Stryer L (2006) Biochemistry 6th edition, W. H. Freeman & Co. ISBN 978-0716767664
T. M. Devlin (2005) Textbook of Biochemistry with Clinical Correlations 6th edition. Wiley Blackwell. ISBN 978-0471678083
Module Learning Strategies
Contact time in the form of 12 x 2-hr lectures will be used to identify and explain the areas of study and to create the framework and guidance for further independent study. The independent study time will be used to carry out short formative tasks to allow guided collation of relevant information to the subject area, as well as to prepare the assignment and revise for the exam.
Module Additional Assessment Details
Students will be required to complete 3 pieces of summative assessment.
20% 45 minute in class unseen test (i)
50% 1.5 hour exam (i, ii)
30% 2000-word essay comparing and contrasting metabolic dysfunction as a result of metabolism or aberrant nutrition of a specific metabolite. (iii)
Students will be provided with formative assessment and feedback via:
Completion of specimen exam questions
Submission of a draft essay
Module Resources
Lecture room
Internet linked PC
Library
Video facilities available in the Science LRC.
Module Indicative Content
Within this module you will study:
- the core pathways of metabolism involved in respiration and biosynthesis of major classes of molecules and of the mechanisms used to control them
- the adaptation of human metabolism to nutritional intake and the relevance of micronutrients to the function of metabolic reactions
- the consequences of derangements within human metabolic pathways as a result of inborn errors and how these may be controlled